By Derrick Lonsdale
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Extra resources for A Nutritionist's Guide to the Clinical Use of Vitamin B-1
Indeed, one of the radical changes that must be made in such an ideological context is that there is a distinct difference between psychosomatic disease ("loss of ease") and malingering. Therefore, our interest has been generated by accepting that if the patient expresses a given symptom — pain for example — there must be a mechanism in terms of neurological and biological transmission. '' In a sense this is true, since the brain is receiving distress signals which throw no light on the site of somatic reference by clinical examination.
Further examples of unusual family histories are described in Chapter 5. The cases reported here are in order to illustrate the diversity of autonomic dysfunction, and to show that it might be a key phenomenon in sudden death. 23 III. 8 FIGURE 4 Hypertension. Recurrent "Hypoglycemic Coma" Twins, died in infancy. "Blue Babies" Epilepsy; Under psychiatric treatment SIDS age 4 months: Autopsy Recurrent Urinary Tract Infections: Renal Calculi Stillbirth Died in infancy. "Blue Baby" Lupus Arthritis "Multiple Sclerosis and Hodgkin's Disease" Died, age 2 days.
This particular laboratory test is discussed in detail in the next chapter, but it has been shown that an accelerated uptake of TPP by red cells is clearly indicative of deficiency of vitamin B1(thiamin) in the cells being tested. Our experience indicates that this test has far-reaching implications, and that it is by no means a specific indicator of nutritional deficiency. In a patient with trisomy 21 and sleep apnea, in another patient with Prader Willi syndrome dying of cor pulmonale, and a boy who had an epileptic fit in a swimming pool and suffered prolonged hypoxia of the brain, we have seen a similar effect upon this laboratory test.
A Nutritionist's Guide to the Clinical Use of Vitamin B-1 by Derrick Lonsdale